Intro
This course illustrates the foundational underpinnings of pain, pain assessment and pain management strategies using a longitudinal case of acute to persistent/chronic orofacial pain with mixed nociceptive and neuropathic phenotypes.
It will provide a common basis for different healthcare professionals to learn the same language and have a shared understanding of pain mechanisms and major biopsychosocial concepts important to the care of persons with pain.
Learning Objectives
- Explain the physiological mechanisms of pain (the biological contribution to pain in the biopsychosocial model)
- Describe genetic mechanisms of pain susceptibility
- Appreciate the mechanisms that modulate acute and chronic pain susceptibility (psychosocial of the biopsychosocial model)
- Apply knowledge of the physiological and genetic mechanisms of pain and pain susceptibility, and mechanisms that modulate acute and chronic pain susceptibility in case presentations, patient simulations, and care of patients
- Develop differential diagnosis based on potential mechanisms of pain given patient’s self-reported assessment data.
- Explain the physiological mechanisms of pain (the biological contribution to pain in the biopsycosocial model).
- Describe the historical development of pain theories and basis for current understanding of pain.
- Explain the physiological mechanisms of pain
- Consider mechanisms that may contribute to the transition from acute to chronic pain
- Apply knowledge of the physiological mechanisms of pain in a case presentation.
- Use appropriate terminology regarding acute, chronic/persistent, and neuropathic pain conditions, including pain characteristics, patient function, and factors influencing neurophysiology of pain and multiple dimensions of pain.
- Acknowledge that differences in pain sensitivity, expectations, pain responses, treatment and response to pain treatment are most likely multifactorial, the result of complex and dynamic interactions of biologic, psychologic, and sociocultural processes. These include genetic factors which may alter pain sensation, perception and efficacy of medications.
- Recognize disparities in pain experience and treatment, including inappropriate provider biases that may unduly contribute to patient suffering.
- Describe genetic mechanisms of pain susceptibility
- Distinguish heritable factors (genetics and epigenetics)
- Identify the multigenic nature of pain, examples of genetic contributions to pain sensitivity and risk for chronic pain
- Use appropriate terminology regarding acute, chronic/persistent, and neuropathic pain conditions, including pain characteristics, patient function, and factors influencing neurophysiology of pain and multiple dimensions of pain.
- Develop differential diagnosis based on potential mechanisms of pain given patients’ self-reported assessment data.
- Discuss the multidimensional nature of pain and its components, implications for patient-families, and relationship to clinical interventions.
- Recognize disparities in pain experience and treatment, including inappropriate provider biases that may unduly contribute to patient suffering
- Acknowledge that race and ethnic differences in pain sensitivity, expectations, pain responses, treatment and response to pain treatment are most likely multifactorial, the result of complex and dynamic interactions of biologic, psychologic, and sociocultural processes, including genetic factors which may alter pain sensation, perception and efficacy of medications.
- Describe the basis for current understanding of pain, gate control, biopsyschosocial model
- Differentiate acute pain from chronic pain
- Consider mechanisms that may contribute to the transition from acute to chronic pain
Target Audience
The target audience is Healthcare Professional Students in medicine, nursing, pharmacy, physical therapy, psychology, and Healthcare Professionals, Faculty and Clinical continuing education.
